Regular ArticleFailure to Demonstrate Human Papillomavirus DNA in Epithelial Ovarian Cancer by General Primer PCR☆
References (30)
- et al.
High frequency of telomeric associations in human ovarian surface epithelial cells transformed by human papilloma viral oncogenes
Cancer Genet Cytogenet
(1997) - et al.
Detection of human papillomavirus DNA in advanced epithelial ovarian carcinoma
Gynecol Oncol
(1987) - et al.
Human papillomavirus and epithelial ovarian neoplasia
Gynecol Oncol
(1989) - et al.
Squamous intraepithelial neoplasia in an ovarian cyst, cervical intraepithelial neoplasia and human papillomavirus
Hum Pathol
(1995) - et al.
Papillomaviruses and Human Disease
(1987) Human pathogenic papillomaviruses
Curr Top Microbiol Immunol
(1994)Papillomavirusus
(1995)Genital Warts. Human papillomavirus Infection
(1995)Challenges of Modern Medicine
Papillomavirus in Human Pathology
(1995)- et al.
Human Papillomavirus Infections in Dermatovenereology
(1997)
New Developments in Cervical Cancer Screening and Prevention
Human papillomavirus in benign and malignant ovarian and endometrial tissues
Int J Gynecol Pathol
Uterine atypical polypoid adenomyoma and ovarian endometrioid carcinoma. Metastatic disease or dual primers
Int J Gynecol Pathol
Absence of human papillomavirus sequences in ovarian pathologies
J Clin Microbiol
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Human papillomavirus and ovarian cancer (review of literature and meta-analysis)
2021, Infection, Genetics and EvolutionCitation Excerpt :A high variability in the prevalence of the virus from 0% (Kaufman et al., 1987) to 83.3% (McLellan et al., 1990) was shown. In the studies over the next decade, the results also vary in a significant range from 0% (Bosch, 2011; Beckmann et al., 1991; Trottier et al., 1995; Anttilaa et al., 1999; Chen et al., 1999) to 66.7% (Lai et al., 1994). Currently, more sensitive methods are used.
Human papillomavirus infection: Is it associated with epithelial ovarian cancer?
2021, Indian Journal of Medical MicrobiologyThe clearance of oral high-risk human papillomavirus infection is impaired by long-term persistence of cervical human papillomavirus infection
2014, Clinical Microbiology and InfectionOvarian cancer
2014, The LancetPersistent oral human papillomavirus infection is associated with smoking and elevated salivary immunoglobulin G concentration
2014, Journal of Clinical VirologyCitation Excerpt :HPV-DNA was extracted from the scrapings with the high salt method [25]. The first HPV-testing for the presence of any high risk HPVs was performed using nested PCR with MY09/MY11 as external and GP05+/GP06+ as internal primers and hybridisation of the PCR products with a digoxigenin-labelled HR-HPV-oligoprobe cocktail [26,27]. Next, HPV genotyping was performed with a Multimetrix kit® (Multimetrix, Progen Biotechnik GmbH, Heidelberg, Germany) detecting 24 low risk (6, 11, 42, 43, 44, and 70) and high risk (16, 18, 26, 31, 33, 35, 39, 45, 51, 52, 53, 56, 58, 59, 66, 68, 73 and 82) HPV-genotypes.
Oral mucosa as a reservoir of human papillomavirus: Point prevalence, genotype distribution, and incident infections among males in a 7-year prospective study
2012, European UrologyCitation Excerpt :HPV testing was done with a nested polymerase chain reaction (PCR) using MY09 and MY11 as external primers and GP05+/GP06+ as internal primers [21]. The PCR products were first hybridised with a digoxigenin-labelled high-risk HPV oligoprobe cocktail (HPV types 16, 18, 31, 33, 35, 39, 45, 51, 52, 54, 56, and 58) to determine whether the samples were high-risk HPV positive or negative [22]. The PCR products available from previous high-risk HPV positive or negative testing were used for HPV genotyping with the Multimetrix kit (Progen Biotechnik, Heidelberg, Germany).
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Becker, YDarai, G